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1. Kidney Beans
If you eat undercooked or raw kidney beans, the toxin phytohaemagglutinin (a lectin, known to interfere with cellular metabolism) can cause extreme nausea, vomiting, diarrhea, abdominal pain and in some cases you may need to be hospitalized. Sometimes known as Red Kidney Bean Poisoning, this condition is caused by eating raw, soaked kidney beans or beans that have been cooked in a crock pot without being boiled (or heated to a high enough temperature first). In fact, heating kidney beans to 176 degrees F may increase their toxicity five-fold compared to eating the raw, which is why outbreaks have been associated with slow cookers or crockpots. Illness can occur from just four or five undercooked cooked or raw beans. In order to make kidney beans safe for consumption, you must soak them for at least 5 hours, get rid of the water and then boil them briskly in fresh water for at least 10-30 minutes.
Though technically a fungus, many species of wild mushrooms contain poisons that can cause illness ranging from mild to deadly. In some cases, symptoms don’t appear for hours, days or even weeks after the mushroom is eaten, and by that time permanent organ damage may have already occurred. Toxins in wild mushrooms cannot be made “safe” by any from of cooking, freezing or processing, and it can be extremely difficult to discern a poisonous mushroom from a safe one. To be safe, unless you’re a trained expert on mushroom identification, don’t eat any mushrooms you find in the wild.
Corn can be contaminated with aflatoxin, a toxin produced by fungus that can grow on certain foods. Aflatoxins are known to cause cancer as well as liver and immune-system problems. Although a human illness outbreak related to aflatoxin has not been reported in the United States, they have occurred in other countries, and aflatoxin-contaminated pet foods have caused outbreaks and deaths among dogs and cats in the United States. It is, however, difficult to prove that a disease such as cirrhosis of the liver or liver cancer is caused by aflatoxin, even though there is reliable evidence that it is an important danger to public health (particularly when unregulated). Other foods that may contain aflatoxin include peanuts, rice, dried coconut meat, cocoa beans, figs, ginger and nutmeg.
If a potato is green or sprouted, it’s a sign that it contains solanine, a compound that is toxic even in small amounts. Eating a green potato, or potato sprouts, can cause what’s known as potato plant poisoning or solanum tuberosum poisoning, leading to symptoms ranging from diarrhea and vomiting to delirium, paralysis, shock and, in extremely rare cases, death.
The leaves and stems of the tomato plant contain glycoalkaloid, a toxin that can lead to stomach upset, headache and dizziness. Green tomatoes do contain some alkaloid poison as well, but generally in too small of quantities to be dangerous. That said, tea made from tomato leaves should be avoided.
Rhubarb stalks, which are actually stems of a perennial plant, are quite tasty when used in pies or crumbles, but the leaves of this plant are very poisonous. If you eat rhubarb leaves, it can cause vomiting and diarrhea, along with seizures, difficulty breathing, kidney problems, coma and even death due to the oxalic acid salts they contain.
7. Lima Beans
Lima beans contain the deadly poison cyanide, which is produced to prevent predators from eating them. Eating large amounts of raw lima beans may cause violent illness and death, so be sure they are thoroughly cooked (soaked and then boiled in fresh water for at least 10 minutes) before eating.
Parsnips contain naturally occurring chemicals called psoralens, which cause genetic mutation and cancer in animals when exposed to ultraviolet light. These toxins are not destroyed by normal cooking, leading researchers to question whether they may have toxic consequences in humans.[ii]
9. Alfalfa Sprouts
Alfalfa sprouts have made the news many times due to contamination with salmonella and e. coli, however even when not contaminated they contain a natural chemical called canavanine that has been found to cause a lupus-like autoimmune disease in an animal study. There is some evidence that people with lupus may want to avoid alfalfa sprouts as they may aggravate the condition.
Spinach contains compounds called oxalates, which can bind to calcium in your body. If eaten in large quantities, there is some evidence that suggests it may contribute to the formation of kidney stones (most kidney stones in U.S. adults are calcium oxalate stones). However, some believe restricting dietary oxalates will not reduce kidney stone formation. Other vegetables that contain oxalates include Swiss chard, beet greens, okra, parsley, collard greens and leeks.
11. Fava Beans
In people with G6PD deficiency, a hereditary abnormality, eating fava beans (and certain other legumes) may destroy red blood less and cause hemolytic anemia — a condition known as favism. This deficit is most common in people from Africa, followed by those from the Mediterranean and southeast Asia.
Though ordinarily healthy, celery topped the Environmental Working Group’s 2011 list of fruits and vegetables most contaminated with pesticides. Coming in at #2 (apples were #1), celery was found to be highly contaminated and tested positive for 57 different pesticides.[iii] If you’re going to eat celery, buying organic makes sense.
(source: http://www.losethebackpain.com/blog/2012/05/16/dangerous-vegetables/)1. Kidney Beans If you eat undercooked or raw kidney beans, the toxin phytohaemagglutinin (a lectin, known to interfere with cellular metabolism) can cause extreme nausea, vomiting, diarrhea, abdominal pain and in some cases you may need to be hospitalized. Sometimes known as Red Kidney Bean Poisoning, this condition is caused by eating raw, soaked kidney ...
Jaundice, (also known as icterus, attributive adjective: icteric) is a yellowish pigmentation of the skin, the conjunctival membranes over the sclerae (whites of the eyes), and other mucous membranes caused by hyperbilirubinemia (increased levels of bilirubin in the blood). This hyperbilirubinemia subsequently causes increased levels of bilirubin in the extracellular fluids. Typically, the concentration of bilirubin in the plasma must exceed 1.5 mg/dL ( > 35 micromoles/L), three times the usual value of approximately 0.5 mg/dL, for the coloration to be easily visible. Jaundice comes from the French word jaune, meaning yellow.
The conjunctiva of the eye are one of the first tissues to change color as bilirubin levels rise in jaundice. This is sometimes referred to as scleral icterus. However, the sclera themselves are not “icteric” (stained with bile pigment) but rather the conjunctival membranes that overlie them. The yellowing of the “white of the eye” is thus more properly termed conjunctival icterus. The term “icterus” itself is sometimes incorrectly used to refer to jaundice that is noted in the sclera of the eyes, however its more common and more correct meaning is entirely synonymous with jaundice.
It was once believed persons suffering from the medical condition jaundice saw everything as yellow. By extension, the jaundiced eye came to mean a prejudiced view, usually rather negative or critical. Alexander Pope, in “An Essay on Criticism” (1711), wrote: “All seems Infected that th’ Infected spy, As all looks yellow to the Jaundic’d Eye.” Similarly in the mid-19th century the English poet Alfred Lord Tennyson wrote in the poem Locksley Hall: “So I triumphe’d ere my passion sweeping thro’ me left me dry, left me with the palsied heart, and left me with a jaundiced eye.”
When a pathological process interferes with the normal functioning of the metabolism and excretion of bilirubin just described, jaundice may be the result. Jaundice is classified into three categories, depending on which part of the physiological mechanism the pathology affects. The three categories are:
Category Definition Pre-hepatic The pathology is occurring prior to the liver. Hepatic The pathology is located within the liver. Post-Hepatic The pathology is located after the conjugation of bilirubin in the liver.
Pre-hepatic jaundice is caused by anything which causes an increased rate of hemolysis (breakdown of red blood cells). In tropical countries, malaria can cause jaundice in this manner. Certain genetic diseases, such as sickle cell anemia, spherocytosis, thalassemia and glucose 6-phosphate dehydrogenase deficiency can lead to increased red cell lysis and therefore hemolytic jaundice. Commonly, diseases of the kidney, such as hemolytic uremic syndrome, can also lead to coloration. Defects in bilirubin metabolism also present as jaundice, as in Gilbert’s syndrome (a genetic disorder of bilirubin metabolism which can result in mild jaundice, which is found in about 5% of the population)and Crigler-Najjar syndrome.
In jaundice secondary to hemolysis, the increased production of bilirubin, leads to the increased production of urine-urobilinogen. Bilirubin is not usually found in the urine because unconjugated bilirubinn is not water-soluble, so, the combination of increased urine-urobilinogen with no bilirubin(since, unconjugated)in urine is suggestive of hemolytic jaundice.
Laboratory findings include:
- Urine: no bilirubin present, urobilirubin > 2 units (i.e., hemolytic anemia causes increased heme metabolism; exception: infants where gut flora has not developed).
- Serum: increased unconjugated bilirubin.
- Kernicterus is associated with increased unconjugated bilirubin.
Hepatocellular (hepatic) jaundice can be caused by acute hepatitis, hepatotoxicity, and alcoholic liver disease. Cell necrosis reduces the liver’s ability to metabolize and excrete bilirubin leading to a buildup of unconjugated bilirubin in the blood. Other causes include primary biliary cirrhosis leading to an increase in plasma conjugated bilirubin. Jaundice seen in the newborn, known as neonatal jaundice, is common, occurring in almost every newborn as hepatic machinery for the conjugation and excretion of bilirubin does not fully mature until approximately two weeks of age. Rat fever (leptospirosis) can also cause hepatic jaundice. In hepatic jaundice, there is invariably cholestasis.
Laboratory findings depend on the cause of jaundice.
- Urine: Conjugated bilirubin present, urobilirubin > 2 units but variable (except in children). Kernicterus is a condition not associated with increased conjugated bilirubin.
Post-hepatic jaundice, also called obstructive jaundice, is caused by an interruption to the drainage of bile in the biliary system. The most common causes are gallstones in the common bile duct, and pancreatic cancer in the head of the pancreas. Also, a group of parasites known as “liver flukes” can live in the common bile duct, causing obstructive jaundice. Other causes include strictures of the common bile duct, biliary atresia, ductal carcinoma, pancreatitis and pancreatic pseudocysts. A rare cause of obstructive jaundice is Mirizzi’s syndrome.
In complete obstruction of the bile duct, no urobilinogen is found in the urine,since bilirubin has no access to the intestine and its in the intestine that bilirubin gets converted to urobilinogen to be later released into the general circulation. In this case, presence of bilirubin(conjugated) in the urine without urine-urobilinogen suggests obstructive jaundice, either intra-hepatic or post-hepatic.
The presence of pale stools and dark urine suggests an obstructive or post-hepatic cause as normal feces get their color from bile pigments. However, although pale stools and dark urine are a feature of biliary obstruction, they can occur in many intra-hepatic illnesses and are therefore not a reliable clinical feature to distinguish obstruction from hepatic causes of jaundice.
Patients also can present with elevated serum cholesterol, and often complain of severe itching or “pruritus” because of the deposition of bile salts.
No single test can differentiate between various classifications of jaundice. A combination of liver function tests is essential to arrive at a diagnosis.
Table of diagnostic tests Function test Pre-hepatic Jaundice Hepatic Jaundice Post-hepatic Jaundice Total bilirubin Normal / Increased Increased Conjugated bilirubin Normal Increased Increased Unconjugated bilirubin Normal / Increased Increased Normal Urobilinogen Normal / Increased Increased Decreased / Negative Urine Color Dark(urobilinogen) Dark (urobilinogen + conjugated bilirubin) Dark (conjugated bilirubin) Stool Color Normal Pale Alkaline phosphatase levels Normal Increased Alanine transferase and Aspartate transferase levels Increased Conjugated Bilirubin in Urine Not Present Present
(more at: http://en.wikipedia.org/wiki/Jaundice)
- If you can spare five minutes & fifty five seconds to see the following link, you would see something that you may not only have seen before but not even imagined.There is an awesome dance called the Thousand-Hand Guanyin, which is making the rounds across the net. Considering the tight coordination required, their accomplishment is nothing short of amazing, even if they were not all deaf.Yes, you read correctly. All 21 of the dancers are complete deaf-mutes.Relying only on signals from trainers at the four corners of the stage, these extraordinary dancers deliver a visual spectacle that is at once intricate and stirring. Its first major international debut was in Athens at the closing ceremonies for the 2004 Paralympics.But it had long been in the repertoire of the Chinese Disabled People’s Performing Art Troupe and had traveled to more than 40 countries. Its lead dancer is 29 year old Tai Lihua, who has a BA from the Hubei Fine Arts Institute.The video was recorded in Beijing during the Spring Festival this year.
Two Heads on same side
Heads on Opposite sides
Two Heads on same side
Two Heads on same side
An aquarium in East Norriton, Pennsylvania, displayed a red-earned slider turtle that had two heads. The reptilian oddity had a pair of front feet on each side, but just one pair of back feet and only one tail.
- Video – Hungarian Jewish
This is the story of a Hungarian Jewish woman who survived Auschwitz and found a coat belonging to a guard which she took to shield her from the cold immediately after her liberation. In the pocket of this coat she found a photo album. It contained pictures of what went on in this extermination camp. Imagine her reaction when she saw a picture of herself coming off of the train as well pictures of her family who were already murdered. This album at Yad Vashem in Jerusalem was donated by this woman in 1980 and will forever be displayed there. When you have 5 minutes of peace and quiet in front of your computer, watch it and consider passing it around to people that you know so they can share it and know about it. It is truly moving and important.
(source:http://www1.yadvashem.org/exhibitions/album_Auschwitz/mutimedia/index.HTML)This is the story of a Hungarian Jewish woman who survived Auschwitz and found a coat belonging to a guard which she took to shield her from the cold immediately after her liberation. In the pocket of this coat she found a photo album. It contained pictures of what went on in this extermination camp. ...